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NF-B, the master transcriptional regulator of pro-inflammatory gene expression, is activated downstream of TNF receptor and IL-1 receptor signaling

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Conclusion In sum, perturbation of Met metabolism may promote the damage process of hepatobiliary injury triggered by viral infections/toxins and/or niche abnormalities, with dysregulated inflammatory and immune responses of the liver, and contribute to the obliteration and eventual fibrosis and atresia of the bile ducts in the initiation/progression of BA